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These alterations collectively induce ventricular electrophysiological instability, characterized by a shortened effective refractory period, prolonged sinoatrial node recovery time, and disrupted myocardial electrical activity and rhythm (Fu et al., 2023)

Upon stimulation by agents such as VEGF, the binding of eNOS and Cav-1 is released, eNOS associates with calmodulin, and then subsequently phosphorylated by heat shock protein 90 and AKT 23

Javitt DC, Zukin SR, Heresco-Levy U, Umbricht D

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